Updated on 2025/06/24

写真a

 
YASUKOCHI TOMOYO
 
Organization
Faculty of Dental Science Department of Dental Science Associate Professor
Graduate School of Dental Science Department of Dental Science（Concurrent）
Graduate School of Dental Science （Concurrent）
School of Dentistry Department of Dentistry（Concurrent）
Title
Associate Professor
Contact information
メールアドレス
Profile
学部教育では、口腔生化学および歯科薬理学を担当している。 　大学院教育および研究活動としては、主に、エピゲノム制御の観点から、周産期や各ライフステージにおける栄養素摂取と疾患罹患感受性の分子基盤に焦点を当てた研究を拡げ、がんを含めた生活習慣病や免疫疾患におけるエピゲノム制御機構、予防や治療に向けた栄養学的アプローチに取り組んでいる。さらに、各種疾患の病態や治療におけるエピゲノム制御機構、個体の代謝制御におけるTCA回路の役割について、その分子基盤を明らかにすることを目指して活動を行っている。
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Research Areas

  • Life Science / Oral pathobiological science

  • Life Science / Oral biological science

  • Life Science / Clinical pharmacy

Degree

  • D.D.S., Ph.D.

Research History

  • Kyushu University 歯学研究院OBT研究センター Associate Professor (In charge of graduate school) 

    2020.3 - Present

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  • Fukuoka University 薬学部 Associate Professor 

    2019.4 - 2020.2

  • Fukuoka University 薬学部 Lecturer 

    2014.10 - 2019.3

  • Kyushu University 大学院歯学研究院 Academic Researcher 

    2013.5 - 2014.9

  • Karolinska Institute, Sweden Department of Women's and Children's Health 日本学術振興会海外特別研究員 

    2012.4 - 2013.4

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    Country：Sweden

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Education

  • Kyushu University   大学院歯学府   歯学専攻

    2003.4 - 2007.3

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    Country：Japan

  • Kyushu University   歯学部   歯学科

    1997.4 - 2003.3

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    Country：Japan

Research Interests・Research Keywords

  • Research theme： The role of Id4 on energy metabolism and epigenomic regulation

    Keyword： Id4, energy metabolism, epigenomics

    Research period： 2021.4 - 2030.3

  • Research theme： Analysis of TCA cycle by-path and itaconic acid in homeostasis regulation

    Keyword： itaconic acid, TCA cycle

    Research period： 2021.4 - 2030.3

  • Research theme： Molecular basis of predisposition to develop non-communicable diseases based on epigenetic modification in the womb

    Keyword： DOHaD, nutrition, non-communicable disease

    Research period： 2015.4 - 2030.3

  • Research theme： Identification of tissue-specific non-coding RNA networks in autoimmune disorder

    Keyword： non-coding RNA, autoimmune disorder

    Research period： 2015.4 - 2025.3

  • Research theme： Identification of tissue-specific non-coding RNA networks in cancer progression

    Keyword： non-coding RNA, cancer

    Research period： 2015.4 - 2025.3

  • Research theme： Analysis of molecular basis of biological defense system regulation through protease reaction

    Keyword： protease

    Research period： 2003.4 - 2027.3

Awards

  • 優秀研究者賞

    2025.3   公益財団法人新日本先進医療研究財団  

    安河内（川久保）友世

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    Award type：International academic award (Japan or overseas) 

    令和4年度研究助成金受領者の中で、優れた業績を発表している研究者に対して授与される賞である。

  • The 28th Federation Meeting of Korean Basic Medical Scientists 2021, Best Poster Award

    2021.6   The 28th Federation Meeting of Korean Basic Medical Scientists 2021  

  • 第6回ロート女性健康科学研究賞

    2015.5  

  • 第22回歯科基礎医学会賞（薬理学部門）

    2010.9   歯科基礎医学会  

  • The 6th General Meeting of the International Proteolysis Society, Travel Award

    2009.10   International Proteolysis Society  

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Papers

  • 4-Octyl Itaconate Attenuates Cell Proliferation by Cellular Senescence <i>via</i> Glutathione Metabolism Disorders and Mitochondrial Dysfunction in Melanoma Reviewed International coauthorship

    Hayashi Y., Saeki A., Yoshimoto S., Yano E., Yasukochi A., Kimura S., Utsunomiya T., Minami K., Aso Y., Hatakeyama Y., Lo Y.C., Hirata M., Jimi E., Kawakubo-Yasukochi T.

    Antioxidants &amp; Redox Signaling   42 ( 10-12 )   547 - 565   2025.2   （ ISSN:1523-0864 eISSN:1557-7716 ）

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    Authorship：Last author,　Corresponding author   Publishing type：Research paper (scientific journal)   Publisher：Mary Ann Liebert Inc  

    Aims: Itaconate (IA) is synthesized in the citric acid cycle via cis-aconitate decarboxylase (ACOD1); however, its biological significance in cancer remains incompletely understood. In previous studies, 4-octyl itaconate (OI) was used as a membrane-permeable form of IA, but little detailed verification of the difference in biological activities between IA and OI exists. Here, we investigated the direct effects of IA and OI on melanoma. Results: The proliferation of melanoma cells treated with OI was significantly suppressed in vitro, and our transcriptomic analysis revealed drastic changes in the expression of glutathione metabolism-related genes in OI-treated cells. Indeed, OI treatment decreased intracellular glutathione levels, followed by increased production of reactive oxygen species and expression of γH2AX, a marker of DNA damage, and β-galactosidase, a marker of cellular senescence. We further showed that the mitochondrial respiratory capacity in B16 cells was significantly decreased by OI treatment. OI administration also suppressed the growth of B16 tumor transplants in vivo, and the expression of γH2AX was increased in tumor tissues of OI-treated mice. In addition, minimal effects of OI treatment were observed in melanocytes and normal tissues. We also proved that not only exogenous IA, which enters intracellularly, but also endogenous IA has little effect on melanoma proliferation activity, via an investigation using Acod1-overexpressing transfectants and Acod1-deficient mice. Conclusion: This work revealed that OI disrupts the antioxidant system via the collapse of glutathione metabolism and inhibits cancer cell proliferation. Antioxid. Redox Signal. 42, 547-565.

    DOI： 10.1089/ars.2024.0629

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    Other Link： https://www.liebertpub.com/doi/pdf/10.1089/ars.2024.0629

  • The impact of environmental enrichment on energy metabolism in ovariectomized mice. Reviewed International coauthorship International journal

    Chaoran Ju, Ayano Ogura, Yoshikazu Hayashi, Yuko Kawabata, Fulvio D'Acquisto, Tomoyo Kawakubo-Yasukochi, Eijiro Jimi

    PloS one   20 ( 4 )   e0320180   2025

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    Language：English   Publishing type：Research paper (scientific journal)   Publisher：Plos One  

    After menopause, a decline in ovarian function leads to various physical and psychological changes, potentially resulting in a range of pathological conditions, including abnormalities in energy metabolism. In recent years, environmental enrichment, which is characterized by positive and comfortable eustress, has been shown to improve various physiological and pathological conditions. This study investigated the effects of environmental factors on energy metabolism in a menopause model using an ovariectomized (OVX) mouse model. Wild-type female mice (8-week-old) were subjected to OVX or a sham operation and maintained under standard condition (SC), enriched environment (EE), or isolated (IS) condition for 4 weeks. OVX led to weight gain and disruption of circadian rhythms, along with changes in various metabolic parameters influenced by differences in housing environments; i.e., EE improved metabolic parameters, but IS deteriorated them. Physical activity and social interaction were factors that determined these differences. Menopause is usually a significant transitional period in a woman's life, and changes in the social environment during this period can contribute to a diverse range of physical and psychological symptoms. Consequently, when implementing interventions to alleviate menopause-related pathological conditions, not only physical symptoms but also the social context should be carefully considered.

    DOI： 10.1371/journal.pone.0320180

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  • Activation of NF-κB signaling regulates ovariectomy-induced bone loss and weight gain. Reviewed International journal

    Fei Huang, Jing Gao, Aonan Li, Akiko Mizokami, Miho Matsuda, Kazuhiro Aoki, Takenobu Katagiri, Tomoyo Kawakubo-Yasukochi, Eijiro Jimi

    Biochimica et biophysica acta. Molecular basis of disease   1870 ( 7 )   167320 - 167320   2024.6   （ ISSN:09254439 ）

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    Language：English   Publishing type：Research paper (scientific journal)   Publisher：Biochimica et Biophysica Acta - Molecular Basis of Disease  

    Postmenopausal women experience bone loss and weight gain. To date, crosstalk between estrogen receptor signals and nuclear factor-κB (NF-κB) has been reported, and estrogen depletion enhances bone resorption by osteoclasts via NF-κB activation. However, it is unclear when and in which tissues NF-κB is activated after menopause, and how NF-κB acts as a common signaling molecule for postmenopausal weight gain and bone loss. Therefore, we examined the role of NF-κB in bone and energy metabolism following menopause. NF-κB reporter mice, which can be used to measure NF-κB activation in vivo, were ovariectomized (OVX) and the luminescence intensity after OVX increased in the metaphyses of the long bones and perigonadal white adipose tissue, but not in the other tissues. OVX was performed on wild-type (WT) and p65 mutant knock-in (S534A) mice, whose mutation enhances the transcriptional activity of NF-κB. Weight gain with worsening glucose tolerance was significant in S534A mice after OVX compared with those of WT mice. The bone density of the sham group in WT or S534A mice did not change, whereas in the S534A-OVX group it significantly decreased due to the suppression of bone formation and increase in bone marrow adipocytes. Disulfiram, an anti-alcoholic drug, suppressed OVX-induced activation of NF-κB in the metaphyses of long bones and white adipose tissue (WAT), as well as weight gain and bone loss. Overall, the activation of NF-κB in the metaphyses of long bones and WAT after OVX regulates post-OVX weight gain and bone loss.

    DOI： 10.1016/j.bbadis.2024.167320

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  • Effects of Maternal Nutrition on Oral Health in Offspring Invited Reviewed

    Kawakubo-Yasukochi T, Hayashi Y, Hirata M

    Current Oral Health Reports   10 ( 3 )   69 - 74   2023.7   （ eISSN:2196-3002 ）

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    Authorship：Lead author,　Corresponding author   Publishing type：Research paper (scientific journal)   Publisher：Springer Science and Business Media LLC  

    Purpose of Review: According to the Developmental Origins of Health and Disease (DOHaD) theory, nutritional status in utero can permanently change body structure, function, and metabolism and eventually regulate the susceptibilities toward various diseases in later life. This review aims to examine and summarize the findings of previous reports, revealing how maternal nutrition during pregnancy and lactation affects oral health in offspring. Recent Findings: A limited number of studies have found an evident relationship between maternal nutrition and children’s oral health. Some evidence suggests that maternal nutrients such as sucrose, proteins, minerals, and vitamins are associated with oral and maxillofacial development, including bone and tooth size, and the risk of dental caries, periodontal disease, and oral cancer in offspring. Summary: The lessons of DOHaD theory from the perspective of the oral health of offspring reveal how in utero nutrients affect not only oral health but also total health throughout life. Oral disease and dysfunction can be prevented in the next generation by improving maternal nutrition during pregnancy and lactation periods.

    DOI： 10.1007/s40496-023-00338-z

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    Other Link： https://link.springer.com/article/10.1007/s40496-023-00338-z/fulltext.html

  • Id4 modulates salivary gland homeostasis and its expression is downregulated in IgG4- related disease via miR-486-5p Reviewed International journal

    Hayashi Y, Kimura S, Yano E, Yoshimoto S, Saeki A, Yasukochi A, Hatakeyama Y, Moriyama M, Nakamura S, Jimi E, Kawakubo-Yasukochi T.

    Biochemical et Biophysics Acta (BBA) - Molecular Cell Research   119404 ( 2 )   119404 - 119404   2023.2   （ ISSN:0167-4889 ）

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    Language：English   Publishing type：Research paper (scientific journal)   Publisher：Elsevier BV  

    Salivary glands are physiologically orchestrated by the coordinated balance between cell differentiation, proliferation, apoptosis, and interactions between epithelial, mesenchymal endothelial, and neuronal cells, and they are frequent sites of manifestations of Sjögren's syndrome (SS) or IgG4-related disease (IgG4-RD). However, little is known about salivary gland homeostasis and its involvement in those diseases. Inhibitor of DNA binding/differentiation 4 (Id4) is an Id protein involved in the transcriptional control of many biological events, including differentiation. Studies of Id4-deficient mice revealed that Id4-deficient submandibular glands were smaller and exhibited accelerated differentiation, compared with those from wild-type littermates. In addition, dry mouth symptoms and Th17 expansion in splenocytes were also observed in the absence of Id4. Furthermore, Id4 levels in the salivary glands of patients with IgG4-RD, but not SS, were significantly decreased compared with those of healthy controls. miRNA-mRNA integrated analysis demonstrated that miR-486-5p was upregulated in IgG4-RD patients and that it might regulate Id4 in the lesion sites. Together, these results provide evidence for the inhibitory role of Id4 in salivary differentiation, and a critical association between Id4 downregulation and IgG4-RD.

    DOI： 10.1016/j.bbamcr.2022.119404

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Presentations

  • 4-octyl itaconate induces cellular senescence in cancer cells

    Hayashi Y, Saeki A, Hirata M, Jimi E, Kawakubo-Yasukochi T.

    第130回日本解剖学会/第102回日本生理学会/第98回日本薬理学会合同大会  2025.3 

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    Event date： 2025.3

    Language：English   Presentation type：Poster presentation  

  • The role of Id4 in salivary gland homeostasis

    Kimura S, Hayashi Y, Yano E, Yoshimoto S, Saeki A, Yasukochi A, Hirata M, Jimi E, Kawakubo-Yasukochi T.

    the 25th Korea-Japan Joint Seminar on Pharmacology  2024.11 

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    Event date： 2024.11

    Language：English   Presentation type：Poster presentation  

    Venue：Jeju, Korea  

  • Itaconate and itaconate analog: Antitumor activity in melanoma

    Saeki A, Hayashi Y, Yoshimoto S, Kimura S, Hirata M, Jimi E, Kawakubo-Yasukochi T.

    the 25th Korea-Japan Joint Seminar on Pharmacology  2024.11 

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    Event date： 2024.11

    Language：English   Presentation type：Poster presentation  

    Venue：Jeju, Korea  

  • 胎児期栄養環境と成熟後エネルギー代謝異常の因果メカニズムの追究 Invited

    安河内（川久保）友世、自見英治郎、平田雅人

    第66回歯科基礎医学会学術大会  2024.11 

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    Event date： 2024.11

    Language：Japanese   Presentation type：Symposium, workshop panel (nominated)  

    Venue：長崎  

  • イタコン酸を用いたがん進展制御へのアプローチ

    林慶和、佐伯彩華、平田雅人、自見英治郎、安河内（川久保）友世

    第66回歯科基礎医学会学術大会  2024.11 

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    Event date： 2024.11

    Language：Japanese   Presentation type：Symposium, workshop panel (public)  

    Venue：長崎  

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MISC

  • Effects of Maternal Nutrition on Oral Health in Offspring Reviewed

    Kawakubo-Yasukochi T, Hayashi Y, Hirata M.

    Current Oral Health Reports (accepted)   2023.6

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    Language：English   Publishing type：Article, review, commentary, editorial, etc. (scientific journal)  

  • The roles of osteocalcin in lipid metabolism in adipose tissue and liver Reviewed

    Otani T, Mizokami A, @Kawakubo-Yasukochi T, Takeuchi H, Inai T, Hirata M.

    Adv Biol Regul   2020.12

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    Language：English   Publishing type：Article, review, commentary, editorial, etc. (scientific journal)  

    DOI： https://doi.org/10.1016/j.jbior.2020.100752

  • 次世代肥満を制御する妊娠期オステオカルシンの有用性 Reviewed

    安河内（川久保）友世、溝上顕子、平田雅人

    2017.10

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    Language：Japanese   Publishing type：Article, review, commentary, editorial, etc. (scientific journal)  

  • 骨基質蛋白質による次世代生活習慣病の予防を目指して

    安河内(川久保) 友世、溝上 顕子、平田 雅人

    Medical Science Digest   2017.5

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    Language：Japanese   Publishing type：Article, review, commentary, editorial, etc. (scientific journal)  

  • Osteocalcin and its endocrine functions Reviewed

    Mizokami A, Kawakubo-Yasukochi T, Hirata M.

    Biochem Pharmacol   2017.5

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    Language：English   Publishing type：Article, review, commentary, editorial, etc. (scientific journal)  

    DOI： 10.1016/j.bcp.2017.02.001.

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Industrial property rights

Patent   Number of applications: 8   Number of registrations: 3
Utility model   Number of applications: 0   Number of registrations: 0
Design   Number of applications: 0   Number of registrations: 0
Trademark   Number of applications: 0   Number of registrations: 0

Professional Memberships

  • Japanese Society for Proteases in Pathophysiology

  • Japanese Cancer Association

  • Japanese Association for Oral Biology

  • 日本薬理学会

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  • 日本生化学会

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Committee Memberships

  • 日本生化学会九州支部例会   Councilor   Domestic

    2025.6 - Present   

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    Committee type：Academic society

  • 歯科基礎医学会   代議員、広報委員   Domestic

    2024.4 - Present   

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    Committee type：Academic society

  • 日本薬理学会   国際対応アソシエイツ   Domestic

    2023.4 - Present   

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    Committee type：Academic society

  • 九州大学   男女共同参画推進室室員  

    2023.4 - 2025.3   

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    Committee type：Other

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  • 日本病態プロテアーゼ学会   Councilor   Domestic

    2020.9 - Present   

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Research Projects

  • 胎内栄養環境が規定する成熟後の肥満・異所性脂肪蓄積の素因形成メカニズム

    Grant number：25K02802  2025.4 - 2029.3

    Grants-in-Aid for Scientific Research  Grant-in-Aid for Scientific Research (B)

    安河内 友世

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    Grant type：Scientific research funding

    本研究では、妊娠中に高脂肪高ショ糖食や各種ビタミン欠乏食・過剰食を摂取した母親マウスの産仔について、網羅的解析により得られる DNAメチル化や遺伝子発現変化を統合的に解析することで、疾患感受性遺伝子の同定を行い、それら遺伝子についての機能解析を行う。またその結果に基づき、妊娠母体あるいは臍帯血中の当該栄養素と産子の代謝パラメータとの相関について、ヒトでの検証（出生コホートを用いた解析）を行う。

    CiNii Research

  • 原因不明不育症患者におけるプロゲステロン応答に着目した新たな流産予防戦略の開発

    Grant number：25K12705  2025.4 - 2029.3

    Grants-in-Aid for Scientific Research  Grant-in-Aid for Scientific Research (C)

    後藤 志信, 市川 智子, 杉浦 真弓, 吉原 紘行, 尾崎 康彦, 川久保友世

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    Grant type：Scientific research funding

    本邦では不育症は5%と欧米より高頻度であり、不育症の克服は少子化対策に直結する。25原因不明患者に対し、欧州のRCTで流産回数が多く性器出血を伴う患者へのプロゲステロン（P4）腟錠の有効性が報告されたが、妊娠年齢の高齢化が進んでいる本邦において流産を繰り返す前に治療効果のある患者を選び出し流産を予防する戦略を開発することが重要である。本研究ではP4シグナルに関連する遺伝子多型が不育症病態におけるP4感受性に関わっているとの仮説のもと、P4応答群と不応群とでゲノムワイド関連解析（GWAS）を用いて遺伝子多型を同定する。これらの成果はヒト妊娠維持メカニズムの新たな解明に発展できる。

  • 植物由来天然化合物の探索とその応用

    2024.9 - 2030.3

    エシカルライフ株式会社 

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    Authorship：Principal investigator  Grant type：Joint research

  • Investigation on obesity predisposition and its patho-physiological properties regulated by transgenerational epigenomic regulation

    Grant number：24K02868  2024.4 - 2028.3

    Grants-in-Aid for Scientific Research  Grant-in-Aid for Scientific Research (B)

    平田 雅人, 安河内 友世

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    Grant type：Scientific research funding

    生活習慣病の多くは肥満に起因する。一旦発症すると治療効果が得難いことから、その制圧には肥満の予防策を講じることが重要である。ところが今、肥満予防には、生後の取り組みに加えて新たな視点が必要となっている。
    申請者は現在までに、動物実験で妊娠母体の栄養状態によって次世代の仔に肥満が現れる事を認め、肥満の仔で生じているエピゲノム異常の共通項を肥満感受性エピゲノムとした。そこで本研究では、細胞や動物を用いた生物学実験とヒト疫学研究を組み合わせて母体の栄養（因）と児の肥満発症素因（果）の関係について検証し、肥満感受性や肥満症多様性を決定する世代を超えるエピゲノム変化の同定と検証を行う。

    CiNii Research

  • 女性ホルモン欠乏によるNF-κB活性化機構と骨・エネルギー代謝制御の解明

    Grant number：24K02612  2024.4 - 2027.3

    Grants-in-Aid for Scientific Research  Grant-in-Aid for Scientific Research (B)

    自見 英治郎, 安河内 友世, 片桐 岳信

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    Grant type：Scientific research funding

    閉経後の女性では、骨密度の低下と共に脂肪の増加による体重増加が起こる。我々は卵巣摘出マウスモデルにNF-κB阻害剤を投与すると、骨量の減少が抑制されるだけでなく、体重増加も抑制することを報告した。以上のような学術的背景と独自の研究成果に基づき、骨量減少と体重増加に重要な女性ホルモンの欠乏で起きるNF-κBシグナルの活性化組織・細胞を同定し、特異的にNF-κBシグナルを抑制できれば、閉経後の骨量減少とエネルギー代謝の悪化に対する革新的な治療法や予防法が確立できると考えた。本研究成果は、新たな骨代謝とエネルギー代謝間のクロストークを提唱するだけでなく、国民のQOLの延伸に貢献できる。

    CiNii Research

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Educational Activities

  • 学部２年生に対する口腔生化学の講義の一部と実習を担当。
    学部４年生に対する歯科薬理学の講義の一部（悪性腫瘍）を担当。
    その他、大学院生への研究指導を行っている。

Class subject

  • スタートアップ倫理学

    2023.4 - Present   Full year

  • 口腔脳機能病態学

    2021.4 - Present   Full year

  • 歯科薬理学

    2020.4 - Present   Full year

  • 口腔生化学

    2020.4 - Present   Full year

Visiting, concurrent, or part-time lecturers at other universities, institutions, etc.

  • 2025  香蘭女子短期大学・食物栄養学科・解剖生理生化学実験   Classification:Part-time lecturer  Domestic/International Classification:Japan 

  • 2024  香蘭女子短期大学・食物栄養学科・解剖生理生化学実験  Classification:Part-time lecturer  Domestic/International Classification:Japan 

  • 2023  香蘭女子短期大学・食物栄養学科・解剖生理生化学実験  Classification:Part-time lecturer  Domestic/International Classification:Japan 

  • 2022  博多メディカル専門学校・歯科衛生士科・薬理学  Classification:Part-time lecturer  Domestic/International Classification:Japan 

  • 2022  香蘭女子短期大学・食物栄養学科・解剖生理生化学実験  Classification:Part-time lecturer  Domestic/International Classification:Japan 

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Teaching Student Awards

  • 藤野博賞

    Year and month of award：2025.3

    Classification of award-winning students：Doctoral student   Name of award-winning student：佐伯彩華

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  • 九州大学学術研究活動表彰

    Year and month of award：2025.3

    Classification of award-winning students：Doctoral student   Name of award-winning student：佐伯彩華

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  • Special Poster Presentation Award

    Year and month of award：2024.11

    Classification of award-winning students：Doctoral student   Name of award-winning student：佐伯彩華

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  • 銅賞

    Year and month of award：2024.6

    Classification of award-winning students：Doctoral student   Name of award-winning student：佐伯彩華

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  • 優秀賞

    Year and month of award：2023.9

    Classification of award-winning students：Doctoral student   Name of award-winning student：佐伯彩華

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Other educational activity and Special note

  • 2016  Special Affairs  平成28年度 薬剤師継続学習通信教育 前期スクーリング講座講師（佐賀県薬剤師会）「免疫抑制剤の基礎知識」

    　詳細を見る

    平成28年度 薬剤師継続学習通信教育 前期スクーリング講座講師（佐賀県薬剤師会）「免疫抑制剤の基礎知識」

  • 2016  Special Affairs  第49回薬学部卒後教育講座（福岡大学市民カレッジ）講師「骨代謝からみた生活習慣病」

    　詳細を見る

    第49回薬学部卒後教育講座（福岡大学市民カレッジ）講師「骨代謝からみた生活習慣病」

Outline of Social Contribution and International Cooperation activities

  • International Affair

Social Activities

  • 薬剤師継続学習通信教育講座 平成28年度 前期スクーリング講座

    Role(s)：Lecturer

    一般社団法人 佐賀県薬剤師会男女共同参画委員会  薬剤師継続学習通信教育講座 平成28年度 前期スクーリング講座  佐賀県薬剤師会館  2016.11

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    Type：Lecture

    「免疫抑制剤の基礎知識」

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  • 薬学部卒後教育講座（福岡大学市民カレッジ）

    Role(s)：Lecturer

    福岡大学  薬学部卒後教育講座（福岡大学市民カレッジ）  福岡大学薬学部  2016.10

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    Audience：Researchesrs,　General

    Type：Lecture

    「骨代謝からみた生活習慣病」

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Media Coverage

  • 産経新聞web版びっくりサイエンスの記事に、研究成果が取り上げられた。 Newspaper, magazine

    産経新聞  2021.11

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    産経新聞web版びっくりサイエンスの記事に、研究成果が取り上げられた。

Travel Abroad

  • 2025.1 - 2025.3

    Staying countory name 1：Singapore   Staying institution name 1：シンガポール国立大学

  • 2024.2 - 2024.5

    Staying countory name 1：Singapore   Staying institution name 1：シンガポール国立大学

  • 2023.8 - 2023.11

    Staying countory name 1：Taiwan, Province of China   Staying institution name 1：国立台湾大学

  • 2013.11 - 2015.4

    Staying countory name 1：Sweden   Staying institution name 1：Karolinska Institute

Year of medical license acquisition

  • 2003