Updated on 2024/10/03

Information

 

写真a

 
JINNO TEPPEI
 
Organization
Kyushu University Hospital General Dentistry Assistant Professor
Abolition organization Comprehensive Dentistry(Joint Appointment)
School of Dentistry Department of Dentistry(Joint Appointment)
Graduate School of Dental Science (Joint Appointment)
Graduate School of Dental Science Department of Dental Science(Joint Appointment)
Title
Assistant Professor
Contact information
メールアドレス
Tel
0926426490
Profile
研究:口腔扁平上皮癌におけるIL-6の発現と機能に関する研究 : 特に抗癌剤抵抗性との関連について研究を遂行している。 臨床:口腔総合診療科にて口腔総合診療、特に口腔外科を専門として外来診療を行っている。また周術期患者の口腔機能管理を担当している。 教育:口腔総合診療科にて歯科医師の卒後研修指導を行っている。学部学生の講義および臨床実習を担当している。
External link

Research Interests・Research Keywords

  • Research theme:Elucidation of risk factors of oral status related to the development of delirium in ICU patients

    Keyword:Oral flora、tongue bacterial count、ICU patients、delirium

    Research period: 2023.4

Awards

  • 優秀ポスター賞

    2014.5   口腔科学会   口腔扁平上皮癌における IL-6 の発現と機能〜抗癌剤抵抗性獲得への関与について〜

Papers

  • Significant association of increased PD-L1 and PD-1 expression with nodal metastasis and a poor prognosis in oral squamous cell carcinoma. Reviewed International journal

    Maruse Y, Kawano S, Jinno T, Matsubara R, Goto Y, Kaneko N, Sakamoto T, Hashiguchi Y, Moriyama M, Toyoshima T, Kitamura R, Tanaka H, Oobu K, Kiyoshima T, Nakamura S.

    International Journal of Oral and Maxillofacial Surgery   47 ( 7 )   836 - 845   2018.7

     More details

    Language:English   Publishing type:Research paper (scientific journal)  

    DOI: 10.1016/j.ijom.2018.01.004

  • Cytokeratin 17 mRNA as a prognostic marker of oral squamous cell carcinoma. Reviewed International journal

    Kitamura R, Toyoshima T, Tanaka H, Kawano S, Matsubara R, Goto Y, Jinno T, Maruse Y, Oobu K, Nakamura S.

    Oncology Letters   14 ( 6 )   6735 - 6743   2017.12

     More details

    Language:English   Publishing type:Research paper (scientific journal)  

    DOI: 10.3892/ol.2017.7066

  • Differential roles of kallikrein-related peptidase 6 in malignant transformation and ΔNp63β- mediated epithelial-mesenchymal transition of oral squamous cell carcinoma. Reviewed International journal

    75   148 - 157   2017.12

     More details

    Language:English   Publishing type:Research paper (scientific journal)  

    We previously reported that epithelial-to-mesenchymal transition (EMT) was mediated by ΔNp63β in oral squamous cell carcinoma (OSCC). In this study, DNA microarray analyses were performed using ΔNp63β-overexpressing OSCC cells to identify genes associated with ΔNp63β-mediated EMT. Thereby, we focused on kallikrein-related peptidase (KLK) 6, most up-regulated following ΔNp63β-overexpression, that activates protease-activated receptors (PARs). In RT-PCR analyses, ΔNp63 was positively associated with KLK6 and PAR2 and negatively with PAR1 in OSCC cells. By ΔNp63 knockdown, KLK6 and PAR2 expression was decreased and PAR1 was increased. Furthermore, KLK6 knockdown led to enhancing migration and invasion, and inhibiting proliferation, suggesting EMT-phenotypes. Although, in the KLK6 or PAR2 knockdown cells, phosphorylation of ERK was reduced, it was restored in the KLK6 knockdown OSCC cells treated with recombinant KLK6 proteins. Immunohistochemistry showed ΔNp63, KLK6, and PAR2 were more strongly expressed in the epithelial dysplasia and central region of OSCC than normal oral epithelium, whereas PAR1 expression was undetectable. Interestingly, at the invasive front of OSCC, ΔNp63, KLK6, and PAR2 were reduced, but PAR1 was elevated. In addition, the OSCC patients with decreasing KLK6 expression at the invasive front had more unfavourable prognosis. These results suggested differential roles of KLK6 in malignant transformation and EMT; high ΔNp63β expression up-regulates KLK6-PAR2 and down-regulates PAR1, inducing malignant transformation in oral epithelium with stimulating proliferation through ERK signal activation. Moreover, KLK6-PAR2 expression is down-regulated and PAR1 is up-regulated when ΔNp63β expression is decreased, leading to EMT with enhancing migration and invasion through ERK signal reduction at the invasive front.

    DOI: 10.1016/j.oraloncology.2017.11.004

  • ΔNp63β-miR-205 axis in epithelial-mesenchymal transition of oral squamous cell carcinoma via targeting ZEB1 and ZEB2. Reviewed International journal

    2017.11

     More details

    Language:English   Publishing type:Research paper (scientific journal)  

    We previously revealed that epithelial-to-mesenchymal transition (EMT) was mediated by ΔNp63β, a splicing variant of ΔNp63, in oral squamous cell carcinoma (OSCC). Recent studies have highlighted the involvement of microRNA (miRNA) in EMT of cancer cells, though the mechanism remains unclear. To identify miRNAs responsible for ΔNp63β-mediated EMT, miRNA microarray analyses were performed by ΔNp63β-overexpression in OSCC cells; SQUU-B, which lacks ΔNp63 expression and displays EMT phenotypes. miRNAs microarray analyses revealed miR-205 was the most up-regulated following ΔNp63β-overexpression. In OSCC cells, miR-205 expression was positively associated with ΔNp63 and negatively with zinc-finger E-box binding homeobox (ZEB) 1 and ZEB2, potential targets of miR-205. miR-205 overexpression by miR-205 mimic transfection into SQUU-B cells led to decreasing ZEB1, ZEB2, and mesenchymal markers, increasing epithelial markers, and reducing cell motilities, suggesting inhibition of EMT phenotype. Interestingly, the results opposite to this phenomenon were obtained by transfection of miR-205 inhibitor into OSCC cells, which express ΔNp63 and miR-205. Furthermore, target protector analyses revealed direct regulation by miR-205 of ZEB1 and ZEB2 expression. These results showed tumor-suppressive roles of ΔNp63β and miR-205 by inhibiting EMT thorough modulating ZEB1 and ZEB2 expression in OSCC.

    DOI: 10.1002/jcp.26267

  • Critical roles of Wnt5a-Ror2 signaling in aggressiveness of tongue squamous cell carcinoma and production of matrix metalloproteinase-2 via ΔNp63β-mediated epithelial-mesenchymal transition. Reviewed International journal

    69   15 - 25   2017.6

     More details

    Language:English   Publishing type:Research paper (scientific journal)  

    OBJECTIVES:
    We previously showed that ΔNp63β, a splicing variant of ΔNp63, mediated EMT and affected cell motility. DNA microarray was thus performed to elucidate the mechanism that ΔNp63β affects cell motility. As the results, Wnt5a was significantly down-regulated by ΔNp63β overexpression in tongue SCC cell line (SQUU-B) with EMT phenotype.
    MATERIALS AND METHODS:
    Seven OSCC cell lines were used. Expression of ΔNp63, Wnt5a, its receptor Ror2, and matrix metalloproteinases (MMPs) were analyzed by RT-PCR, real-time PCR, and western blotting, and gelatin zymography. Furthermore, we examined the effects of siRNA for Wnt5a or Ror2 and recombinant human Wnt5a (rhWnt5a) on motility of tongue SCC cells. Biopsy specimens from tongue SCC patients were used for immunohistochemical staining of Wnt5a and Ror2.
    RESULTS:
    Wnt5a and Ror2 were expressed only in SQUU-B cells without ΔNp63 expression, and negatively associated with ΔNp63 expression in other cells. ΔNp63β overexpression in SQUU-B cells decreased Wnt5a and Ror2 expression. By Wnt5a or Ror2 knockdown, cell motility was remarkably inhibited, but EMT markers expression was unaffected. MMP-2 expression and the activities inversely correlated with ΔNp63 expression, and were inhibited by Wnt5a or Ror2 knockdown. Cell motility and MMP-2 activities were recovered by adding rhWnt5a in the cells with Wnt5a knockdown, but not in those with Ror2 knockdown. Moreover, immunohistochemical analyses in tongue SCC specimens found that high expression of Wnt5a or Ror2 was associated with poorer prognosis.
    CONCLUSION:
    Wnt5a-Ror2 signaling enhanced tongue SCC cell aggressiveness and promoted production of MMP-2 following ΔNp63β-mediated EMT.

    DOI: 10.1016/j.oraloncology.2017.03.019

  • CD163+CD204+ tumor-associated macrophages contribute to T cell regulation via interleukin-10 and PD-L1 production in oral squamous cell carcinoma. Reviewed International journal

    Kubota K, Moriyama M, Furukawa S, Rafiul HASM, Maruse Y, Jinno T, Tanaka A, Ohta M, Ishiguro N, Yamauchi M, Sakamoto M, Maehara T, Hayashida JN, Kawano S, Kiyoshima T, Nakamura S.

    Scientific Reports   7 ( 1 )   1755 - 1755   2017.5

     More details

    Language:English   Publishing type:Research paper (scientific journal)  

    DOI: 10.1038/s41598-017-01661-z

  • 舌下腺に生じた多形腺腫の1例と文献的考察 Reviewed

    金子直樹、松原良太、川野真太郎、見立英史、神野哲平、丸瀬靖之、坂本泰基、橋口有真、清島保、中村誠司

    日本口腔内科学会雑誌   22 ( 2 )   79 - 83   2016.12

     More details

    Language:Japanese   Publishing type:Research paper (scientific journal)  

    多形腺腫は最も高頻度の唾液腺腫瘍であるが、舌下腺に生じるのはまれである。また舌下腺腫瘍の多くは悪性であるため、診断に苦慮することも多い。われわれは舌下腺多形腺腫の1例を経験したので、文献的考察を加え報告する。症例は60歳代男性で、左側口底部の腫瘤を主訴に来院、外科的切除を行い、多形腺腫の診断を得た。術後1年6ヵ月経過した現在、再発は認めていない。舌下腺腫瘍を疑う際は、悪性に準じた対応が重要である。

    DOI: https://doi.org/10.6014/jjsom.22.79

  • Tongue squamous cell carcinoma producing both parathyroid hormone- related protein and granulocyte colony-stimulating factor: a case report and literature review. Reviewed International journal

    Kaneko N, Kawano S, Matsubara R, Goto Y, Jinno T, Maruse Y, Sakamoto T, Hashiguchi Y, Iida M, Nakamura S.

    World Journal of Surgical Oncology   14 ( 1 )   161 - 161   2016.6

     More details

    Language:English   Publishing type:Research paper (scientific journal)  

    DOI: 10.1186/s12957-016-0918-1

  • Clinicopathological evaluation of pre-operative chemoradiotherapy with S-1 as a treatment for locally advanced oral squamous cell carcinoma. Reviewed International journal

    Kawano S., Zheng Y., Oobu K., Matsubara R., Goto Y., Chikui T., Yoshitake T., Kiyoshima T., Jinno T., Maruse Y., Mitate E., Kitamura R., Tanaka H., Toyoshima T., Sugiura T., Nakamura S.

    Oncology Letters   11 ( 5 )   3369 - 3376   2016.5

     More details

    Language:English   Publishing type:Research paper (scientific journal)  

    DOI: 10.3892/ol.2016.4411

  • Increased expression of interleukin-6 predicts poor response to chemoradiotherapy and unfavorable prognosis in oral squamous cell carcinoma. Reviewed International journal

    Jinno T., Kawano S., Maruse Y., Matsubara R., Goto Y., Sakamoto T., Hashiguchi Y., Kaneko N., Tanaka H., Kitamura R., Toyoshima T., Jinno A., Moriyama M., Oobu K., Kiyoshima T., Nakamura S.

    Oncology Reports   33 ( 5 )   2161 - 2168   2015.5

     More details

    Language:English   Publishing type:Research paper (scientific journal)  

    DOI: 10.3892/or.2015.3838

  • Possible involvement of ΔNp63 downregulation in the invasion and metastasis of oral squamous cell carcinoma via induction of a mesenchymal phenotype. Reviewed International journal

    31 ( 3 )   293 - 306   2014.3

     More details

    Language:English   Publishing type:Research paper (scientific journal)  

    Epithelial-to-mesenchymal transition (EMT), an essential developmental program, is involved in tumor progression. ΔNp63, a homolog of p53, is associated with the EMT program, but the detailed mechanism remains to be elucidated. In this study, we investigated the role of ΔNp63 in EMT during progression of oral squamous cell carcinoma (OSCC). Five OSCC cell lines and specimens from 78 patients with OSCC were used. The expressions of ΔNp63, p63α, p63β and epithelial markers (cytokeratins 5 and 14) was detected in the OSCC cells, but not in SQUU-B cells (high metastatic potential). E-cadherin was expressed in all OSCC cells. Mesenchymal markers were strongly expressed in the SQUU-B cells. Knockdown of endogenous ΔNp63 in HSC-2 cells induced morphological changes to the spindle shape, decreased the expression of epithelial markers, increased the expression of mesenchymal markers, increased migration and reduced proliferation. By contrast, SQUU-B cells overexpressing ΔNp63β showed changed their morphology from stromal cell-like to epithelial cells. However, E-cadherin expression was not affected by ΔNp63 knockdown or overexpression. Immunohistochemical staining revealed that cancer cells expressing vimentin were found at the invasive front in the OSCC specimens. The intensity of ΔNp63 expression was also decreased in these cells. Interestingly, the vimentin positivity or decreased intensity of ΔNp63 was positively associated with metastases and poor prognosis in the OSCC patients. These results indicated that ΔNp63 downregulation in cancer cells induces a mesenchymal phenotype that is related to tumor progression of OSCC.

    DOI: 10.1007/s10585-013-9628-z

  • 頸部再発に対し放射線併用超選択的動注化学療法が奏効した舌扁平上皮癌の1例 Reviewed

    松原良太、川野真太郎、清島保、神野哲平、丸瀬靖之、中村誠司

    日本口腔外科学会雑誌   60 ( 2 )   76 - 81   2014.2

     More details

    Language:Japanese   Publishing type:Research paper (scientific journal)  

    40歳代男。左側舌縁部の白斑を自覚したが、疼痛を認めなかったため放置した。同部に疼痛を自覚し、ステロイド軟膏を処方されるも症状は改善しなかった。MRI所見では、左側舌縁部にT1強調画像にて高信号を呈する辺縁不整の腫瘤を認めた。左側舌癌の診断で、静脈内鎮静下に左側舌部分切除術および術中迅速病理検査を施行した。術後の経過は良好で、術後14日目に退院した。術中ならびに術後の病理組織学的診断は中分化型扁平上皮癌であった。その後、造影CTで左側内頸静脈リンパ節領域に転移が疑われるリンパ節を認め、全身麻酔下に左側根治的頸部郭清術変法を施行した。左側顎下部に弾性硬で無痛性、境界不明瞭な腫瘤を認め、高分化型扁平上皮癌の病理組織学的診断を得た。放射線併用超選択的動注化学療法を施行した。動注化学放射線療法後6年経過した現在も腫瘍の再発および転移は認められず、経過良好である。

    DOI: https://doi.org/10.5794/jjoms.60.76

  • Immunohistochemical location of the p75 neurotrophin receptor (p75NTR) in oral leukoplakia and oral squamous cell carcinoma. Reviewed International journal

    Kiyosue T., Kawano S., Matsubara R., Goto Y., Hirano M., Jinno T., Toyoshima T., Kitamura R., Oobu K., Nakamura S.

    International Journal of Clinical Oncology   18 ( 1 )   154 - 163   2013.2

     More details

    Language:English   Publishing type:Research paper (scientific journal)  

    DOI: 10.1007/s10147-011-0358-4

  • Clinical significance of combined assessment of the maximum standardized uptake value of F-18 FDG PET with nodal size in the diagnosis of cervical lymph node metastasis of oral squamous cell carcinoma. Reviewed International journal

    Matsubara R., Kawano S., Chikui T., Kiyosue T., Goto Y., Hirano M., Jinno T., Nagata T., Oobu K., Abe K., Nakamura S.

    Academic Radiology   19 ( 6 )   708 - 717   2012.6

     More details

    Language:English   Publishing type:Research paper (scientific journal)  

    DOI: 10.1016/j.acra.2012.02.009

  • Increased ΔNp63 expression is predictive of malignant transformation in oral epithelial dysplasia and poor prognosis in oral squamous cell carcinoma. Reviewed International journal

    39 ( 6 )   1391 - 1399   2011.12

     More details

    Language:English   Publishing type:Research paper (scientific journal)  

    This study examined immunohistochemical expression of ΔNp63, a keratinocyte stem cell marker, in oral leukoplakia (OL) and oral squamous cell carcinoma (OSCC) and then to elucidate usefulness of ΔNp63 as a marker for diagnosis and prognosis. One-hundred and twelve cases of OL and 81 cases of OSCC were analyzed by immunohistochemical staining for ΔNp63, Ki-67, and cytokeratin 14. These labeling indices (LIs) were calculated, and the association of these LIs with clinicopathologic characteristics in the OL and OSCC was evaluated. In the OL, these LIs increased significantly according to the severity of epithelial dysplasia (p<0.0001). ΔNp63-LI in the OL with malignant transformation was significantly higher than that in the OL without (49.3 vs. 34.2%; p<0.01). In the OSCC, the LIs increased significantly in association with the histologic grade (p<0.0001). A significant difference between the high and low ΔNp63-LI groups was found in the incidence of cervical lymph node and distant metastasis (p<0.05). The prognosis of the high ΔNp63-LI (mean value >73.8%) group is poorer than that of the low ΔNp63-LI (mean value ≤73.8%) group (p<0.05). These results suggested that increased ΔNp63 expression is involved in malignant transformation in epithelial dysplasia and poor prognosis in OSCC.

    DOI: 10.3892/ijo.2011.1151

▼display all

Presentations

  • 脳死肝移植により救命を得た患者の周術期口腔機能管理に難渋した一症例

    有水智香、神野哲平、赤星朋比古、進藤幸之助、高嶋美甫、永田千尋、中村次代、稲井裕子、和田尚久

    第64回秋季日本歯周病学会学術大会  2021.10 

     More details

    Event date: 2021.10

    Language:Japanese  

    Country:Japan  

  • 当科12年間における口腔内多発癌に対する臨床的検討

    白石 由梨香,丸瀬 靖之,川野 真太郎,松原 良太,後藤 雄一,神野 哲平,坂本 泰基, 橋口 有真,金子 直樹,服部 多市,田中 翔一,濱田栄樹,樋渡 萌美,大部 一成,中村 誠司

    第88回日本口腔外科学会九州支部学術集会  2020.8 

     More details

    Event date: 2021.8 - 2020.8

    Language:Japanese  

    Country:Japan  

  • 広範熱傷患者への歯科的介入の検討

    木附智子、石井広太郎、神野哲平、平野奈々美、大山順子、赤星朋比古、和田尚久、森悦秀

    第18回日本口腔ケア学会総会  2021.4 

     More details

    Event date: 2021.4

    Language:Japanese  

    Country:Japan  

  • 九州大学病院救命ICUにおける多職種連携に有効な歯科的情報共有方法の検討

    有水智香、神野哲平、野口英里、桑田睦子、赤星朋比古、柏崎晴彦、和田尚久

    第18回日本口腔ケア学会総会  2021.4 

     More details

    Event date: 2021.4

    Language:Japanese  

    Country:Japan  

  • 当院ICUにおいてOHAT-Jを活用した口腔ケア支援システムの取り組み

    平野 奈々美,神野 哲平,野口 英里,掘 智恵,有水 智香,岡留 朝子,山添 淳一,和田 尚久

    第17回日本口腔ケア学会総会・学術大会  2020.9 

     More details

    Event date: 2020.9

    Language:Japanese  

    Country:Japan  

  • 周術期口腔機能管理患者のセルフケアの状況とその関連要因

    有水 智香,神野 哲平,田上 祐梨,北岡 優衣,白 仁協,倉田 理沙,祐田 明香,平野 奈々美,山添 淳一,柏崎 晴彦,和田 尚久

    第62回秋季日本歯周病学会学術大会  2019.10 

     More details

    Event date: 2019.10

    Language:Japanese  

    Country:Japan  

  • 舌エコーの画像所見による潜在的頸部リンパ節転移の予測

    服部 多市, 川野 真太郎, 三上 友里恵, 筑井 徹, 河津 俊幸, 松原 良太, 後藤 雄一, 金子 直樹, 坂本 泰基, 橋口 有真, 神野 哲平, 丸瀬 靖之, 田中 翔一, 大部 一成, 中村 誠司

    日本口腔科学会  2018.5 

     More details

    Event date: 2018.5

    Language:Japanese   Presentation type:Oral presentation (general)  

    Venue:ウインクあいち   Country:Japan  

  • 骨吸収抑制薬関連顎骨壊死に対する非侵襲的治療

    梯 裕恵, 神野 哲平, 林田 淳之將, 中村 誠司

    日本口腔ケア学会  2018.4 

     More details

    Event date: 2018.4

    Language:Japanese   Presentation type:Oral presentation (general)  

    Venue:福岡国際会議場   Country:Japan  

  • 高齢入院患者に対し歯科摂食嚥下リハチームによる口腔健康管理によって食事内容の改善に寄与した症例

    井上 昂也, 山添 淳一, 荻野 洋一郎, 神野 哲平, 塚本 葉子, 萱野 綾華, 柏崎 晴彦, 竹下 徹, 和田 尚久

    口腔衛生学会雑誌  2024.7  (一社)日本口腔衛生学会

     More details

    Language:Japanese  

  • 当院における周術期食道癌患者に対する歯科摂食嚥下リハビリテーションチームの取り組み

    友岡 祥子, 井上 昂也, 山添 淳一, 荻野 洋一郎, 神野 哲平, 塚本 葉子, 萱野 綾華, 和田 尚久, 柏崎 晴彦

    日本口腔ケア学会雑誌  2024.4  (一社)日本口腔ケア学会

     More details

    Language:Japanese  

▼display all

Professional Memberships

  • 日本摂食嚥下リハビリテーション学会

  • 日本歯科保存学会

  • 日本口腔科学会

  • 日本口腔外科学会

  • 日本補綴歯科学会

Research Projects

  • ICU患者におけるせん妄発症に関連した口腔状態の危険因子の解明

    Grant number:23K09506  2023 - 2025

    日本学術振興会  科学研究費助成事業  基盤研究(C)

    神野 哲平, 赤星 朋比古, 和田 尚久

      More details

    Authorship:Principal investigator  Grant type:Scientific research funding

    せん妄は ICU 患者の予後不良因子の 1つとして知られている。ICUでのせん妄発症は、ICU在室日数および入院期間の延長、医療費の増大、死亡率の上昇、ICU 退室後も続く認知機能の低下などに関連するため、患者のせん妄発症リスクから発症予測を行うことは、重要な視点となりうる。本研究では、せん妄発症に関連する口腔状態の危険因子を解明する。予備研究において、舌細菌数がせん妄の発症予測因子であることが示唆されており、口腔細菌叢とせん妄との関連性についても解析を行う。ICU患者の早期回復と退院後のQOLの向上へ、口腔から始める新たなせん妄対策を提案することを目指す。

    CiNii Research

  • Research on the role and function of zinc transporters in oral squamous cell carcinoma

    Grant number:19K10313  2019 - 2022

    Japan Society for the Promotion of Science  Grants-in-Aid for Scientific Research  Grant-in-Aid for Scientific Research (C)

    Jinno Teppei

      More details

    Authorship:Principal investigator  Grant type:Scientific research funding

    ZIP6 is a zinc transporter regulated by inflammatory cytokines, and ZIP6 in particular has been shown to be an essential factor for EMT, and is a molecule that is attracting attention for its relationship with cancer growth, invasion, and metastasis. ... In fact, it has been reported that breast cancer cells in which ZIP6 expression is suppressed have their proliferation ability and EMT suppressed, so it is expected that detailed analysis of the expression of these zinc transporters will progress. . In this study, we investigated the expression of zinc transporters in OSCC cells, and investigated the effects of zinc transporters on anticancer drug resistance in OSCC cells and their relationship with IL-6 signaling.

    CiNii Research

  • IL-6シグナル伝達経路を介した口腔扁平上皮癌細胞の抗癌剤抵抗性獲得に関する研究

    Grant number:16K20584  2016 - 2017

    科学研究費助成事業  若手研究(A,B)

      More details

    Authorship:Principal investigator  Grant type:Scientific research funding

Class subject

  • 臨床予備実習

    2023.6 - 2023.8   Summer quarter

  • 臨床実習

    2023.4 - 2024.3   Full year

  • リサーチエクスポージャー

    2023.4 - 2023.6   Spring quarter

  • リサーチエクスポージャー

    2022.12 - 2023.2   Winter quarter

  • リサーチエクスポージャー

    2022.10 - 2022.12   Fall quarter

  • リサーチエクスポージャー

    2022.6 - 2022.8   Summer quarter

  • 臨床予備実習

    2022.6 - 2022.8   Summer quarter

  • 臨床実習

    2022.4 - 2023.3   Full year

  • アーリーエクスポージャー

    2022.4 - 2022.6   Spring quarter

  • 臨床実習

    2021.4 - 2022.3   Full year

  • 臨床実習

    2020.4 - 2021.3   Full year

  • 臨床実習

    2019.4 - 2020.3   Full year

  • 臨床実習

    2018.4 - 2019.3   Full year

  • PBLチュートリアル

    2018.4 - 2018.9   First semester

▼display all

Outline of Social Contribution and International Cooperation activities

  • ・卒後臨床研修歯科医の臨床および技工における知識、技術の指導を行うとともに、患者中心の全人的歯科医療を理解し、患者の信頼に応じ得る倫理観を養成するための指導を行い、社会保健に貢献できる歯科医師を育成する。
    ・OSCEのスタッフとして歯学学生の教育に尽力する。
    ・地域社会連携の一環として他施設歯科診療所にて診療を行い、地域歯科保健に貢献する。

Specialized clinical area

  • Biology / Medicine, Dentistry and Pharmacy / Dentistry / Surgical Dentistry

Clinician qualification

  • Certifying physician

    日本口腔外科学会

  • Certifying physician

    日本口腔科学会

Year of medical license acquisition

  • 2010

Notable Clinical Activities

  • 所属診療科の特色として、診療活動とともに卒後研修歯科医の臨床教育指導を行っている。 また、専門分野が口腔外科であるため、研修歯科医への外科処置技術指導も行っている。