||Mitochondria, oxidative stress and heart failure.
||Overexpression of Twinkle, a mitochondria DNA helicase, protects from cardiac remodeling and failure in mice.
||A new drug delivery system using fibronectin-VEGF fusion is more effective and harmless than VEGF alone for the therapeutic neovascularization.
||Overexpression of mitochondrial transcription factor A protects failing myocardium from oxidative stress.
||Modulation of the myocardial redox state by vagal nerve stimulation after experimental myocardial infarction.
||Overexpression of TFAM prevents age-dependent memory impairment in mice.